In the final game before the FIBA World Cup break on Sunday, August 30, 2026, Nneka Ogwumike recorded 23 points and 12 rebounds to lead the Los Angeles Sparks to a 90-74 victory over the Seattle Storm at Climate Pledge Arena in Seattle, Washington, according to game summaries from the WNBA.
Sparks Control Early Quarters With Dominant Run
The Los Angeles Sparks established control immediately, scoring the first 13 points as part of an opening 18-2 run in the first quarter. According to the WNBA, Dearica Hamby’s layup with 5:45 remaining in the first half capped an 11-0 scoring spurt that extended the Sparks’ advantage to 39-20.
Seattle Struggles From Deep and Loses Horston to Injury
The Seattle Storm failed to find offensive rhythm throughout the match, finishing 4 of 22 from 3-point range across the entire game. According to the WNBA, Dominique Malonga recorded 17 points on 8-of-21 shooting alongside 15 rebounds, while Flau’jae Johnson scored 11 of her 19 points in the fourth quarter. Zia Cooke added 14 points by making 6 of 17 field goal attempts. The loss drops Seattle to 1-18 against Western Conference opponents this season, with their sole divisional win coming in an 82-64 victory over Los Angeles on July 6. The Storm also suffered a personnel loss when Jordan Horston fell awkwardly midway through the third quarter, limped to the locker room, and did not return.
Individual Statistics and Upcoming Schedule
Rae Burrell contributed 22 points for the Sparks, and Cameron Brink added 11 points on 3-of-15 shooting as Los Angeles improved its record to 15-25. The Storm fell to 8-32 overall in front of an announced attendance of 14,200 spectators. Officials Angelica Suffren, Marcy Williams, and Genesis Perrymond managed the floor action. Following the FIBA World Cup break, the Sparks are scheduled to visit Dallas on September 17, while the Storm will host Las Vegas on the same date, according to the WNBA.
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- Breakthrough Salk Study Uncovers Mechanism Behind Immunotherapy Resistance: Interferons, Mitochondrial Dysfunction, and PGE2″ Interferons, mitochondrial dysfunction and PGE2: Salk study reveals mechanism behind immunotherapy resistance. Boost its search engine visibility with relevant keywords for maximum impact. Immunotherapy resistance remains one of the biggest hurdles in cancer treatment. According to a recent study published in the journal Nature Communications, scientists at the Salk Institute have made a groundbreaking discovery that sheds light on the underlying mechanisms behind this resistance. The study reveals that interferons, a type of protein that plays a crucial role in the immune system, can contribute to mitochondrial dysfunction in cancer cells. This dysfunction can lead to the production of prostaglandin E2 (PGE2), a molecule that promotes tumor growth and resistance to immunotherapy. In their study, the researchers found that PGE2 production was a key factor in the development of immunotherapy resistance in cancer cells. The team used a combination of experimental and computational models to investigate the relationship between interferons, mitochondrial dysfunction, and PGE2 production. The findings of the study suggest that targeting PGE2 production could be a potential strategy for overcoming immunotherapy resistance. The researchers propose that blocking PGE2 receptors or inhibiting its production could help restore the function of mitochondria in cancer cells, making them more susceptible to immunotherapy. The study’s authors hope that their findings will pave the way for the development of new therapies that can overcome immunotherapy resistance and improve treatment outcomes for cancer patients. Key Takeaways: – Interferons contribute to mitochondrial dysfunction in cancer cells – Mitochondrial dysfunction leads to PGE2 production, promoting tumor growth and resistance to immunotherapy – Targeting PGE2 production could be a potential strategy for overcoming immunotherapy resistance – Restoring mitochondrial function in cancer cells could make them more susceptible to immunotherapy Keywords: immunotherapy resistance, interferons, mitochondrial dysfunction, PGE2, Salk Institute, cancer treatment, breakthrough study, Nature Communications. (archyworldys.com)