Search Continues for Missing Fishing Vessel and Crew in the Black Sea
Search operations are ongoing for a fishing vessel, the BH 8112, that sank near Maslen Nos in the Black Sea on February 18th. As of February 24, 2026, the bodies of the crew members remain missing, according to the Ministry of Defense.
Vessel Inspection Completed
The Ministry of Defense reports that the inspection of the sunken fishing vessel has been completed by Navy specialists. The investigation confirmed the hull of the BH 8112 is intact, with no evidence of breaches or impacts.
Investigation Underway
All materials gathered during the inspections have been handed over to the National Air, Water and Rail Accident Investigation Board for further analysis. The cause of the sinking remains under investigation.
Search Efforts
Diving teams from the Flotilla of Combat and Auxiliary Ships, the Varna Naval Base, and the Burgas Naval Base participated in the vessel inspection. A remotely controlled underwater device, the SRS Fusion, was also utilized during the search.
Diesel Stain Detected
A stain of diesel fuel has been detected near Maslen Nos, the location where the vessel disappeared, indicating a potential leak from the BH 8112.
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- Breakthrough Salk Study Uncovers Mechanism Behind Immunotherapy Resistance: Interferons, Mitochondrial Dysfunction, and PGE2″ Interferons, mitochondrial dysfunction and PGE2: Salk study reveals mechanism behind immunotherapy resistance. Boost its search engine visibility with relevant keywords for maximum impact. Immunotherapy resistance remains one of the biggest hurdles in cancer treatment. According to a recent study published in the journal Nature Communications, scientists at the Salk Institute have made a groundbreaking discovery that sheds light on the underlying mechanisms behind this resistance. The study reveals that interferons, a type of protein that plays a crucial role in the immune system, can contribute to mitochondrial dysfunction in cancer cells. This dysfunction can lead to the production of prostaglandin E2 (PGE2), a molecule that promotes tumor growth and resistance to immunotherapy. In their study, the researchers found that PGE2 production was a key factor in the development of immunotherapy resistance in cancer cells. The team used a combination of experimental and computational models to investigate the relationship between interferons, mitochondrial dysfunction, and PGE2 production. The findings of the study suggest that targeting PGE2 production could be a potential strategy for overcoming immunotherapy resistance. The researchers propose that blocking PGE2 receptors or inhibiting its production could help restore the function of mitochondria in cancer cells, making them more susceptible to immunotherapy. The study’s authors hope that their findings will pave the way for the development of new therapies that can overcome immunotherapy resistance and improve treatment outcomes for cancer patients. Key Takeaways: – Interferons contribute to mitochondrial dysfunction in cancer cells – Mitochondrial dysfunction leads to PGE2 production, promoting tumor growth and resistance to immunotherapy – Targeting PGE2 production could be a potential strategy for overcoming immunotherapy resistance – Restoring mitochondrial function in cancer cells could make them more susceptible to immunotherapy Keywords: immunotherapy resistance, interferons, mitochondrial dysfunction, PGE2, Salk Institute, cancer treatment, breakthrough study, Nature Communications. (archyworldys.com)